大咖云集！Neuroscience Bulletin诚邀您参加国际疼痛会议
分享近期与疼痛领域相关的国际性会议，这两次大会皆有Neuroscience Bulletin的身影，期待与您的美丽邂逅！
世界疼痛大会（The World Congress on Pain）
世界疼痛大会国际是世界疼痛组织（International Association for the Study of Pain, IASP）的年度品牌会议，是一项具有全球影响力的的国际会议，致力于分享疼痛领域相关研究、治疗和教育方面的新进展。会议汇集了来自100多个国家的数千名代表，涉及多个学科。参与者包括致力于疼痛领域的基础研究人员、临床医生、学生和受训人员。
会议地点：美国，波士顿
会议时间：2018 年9月12日-16日
会议网址：https://www.iaspworldcongressonpain.org/registration/?utm_source=iaspwebsite&utm_medium=homepagecarousel&utm_campaign=earlyregisteration
国际疼痛环路与医学研讨会（The International Symposium for Pain Circuitry and Medicine）
国际疼痛环路与医学研讨会由上海市神经科学学会、浙江省疼痛医学会和上海医学会疼痛医学分会和中国神经科学学会组织举办。本次研讨会将介绍疼痛环路和医学领域的最新进展，弥合基础研究与临床应用之间的差距。会议的初衷是将疼痛领域的学者和临床医生聚集在一起，破解疼痛研究的新思路和新疗法。
会议地点：中国，杭州市
会议时间：2018 年9月20日-22日
会议网址：
http://ispcm.ssn.org.cn/hotels.php
为了更好地了解疼痛领域的最新进展，Neuroscience Bulletin曾在2018年第1期刊发了“痛与痒”专辑（详见征服“痛与痒”——“羿射九日”），我们撷选了15篇发表在2017-2018年Neuroscience Bulletin上疼痛的研究论文和综述（扫描二维码阅读或下载），欢迎下载浏览并引用。
[1] Zhou GK, Jiao YL, Zhou Y, Qin SC, Tao J, Jiang F, et al. Up-Regulation of Akt and Nav1.8 in BmK I-Induced Pain.Neuroscience Bulletin 2018, 34: 539-542.
[2] Ji RR. Recent Progress in Understanding the Mechanisms of Pain and Itch: the Second Special Issue. Neuroscience Bulletin 2018, 34: 1-3.
[3] Reddan MC,Wager TD. Modeling Pain Using fMRI: From Regions to Biomarkers. Neuroscience Bulletin 2018, 34: 208-215.
[4] Chiu IM.Infection, Pain, and Itch. Neuroscience Bulletin 2018, 34: 109-119.
[5] Bang SS,Yoo J, Gong XR, Liu D, Han QJ, Luo X, et al. Differential Inhibition of Nav1.7 and Neuropathic Pain by Hybridoma-Produced and Recombinant Monoclonal Antibodies that Target Nav1.7.Neuroscience Bulletin 2018, 34: 22-41.
[6] Chen LM.Cortical Representation of Pain and Touch: Evidence from Combined Functional Neuroimaging and Electrophysiology in Non-human Primates. Neuroscience Bulletin 2018, 34: 165-177.
[7] Li AL,Zhang JD, Xie WR, Strong JA, Zhang JM. Inflammatory Changes in Paravertebral Sympathetic Ganglia in Two Rat Pain Models. Neuroscience Bulletin 2018, 34:85-97.
[8] Tsuda M.Modulation of Pain and Itch by Spinal Glia. Neuroscience Bulletin 2018, 34:178-185.
[9] Chen G,Luo X, Qadri MY, Berta T, Ji RR. Sex-Dependent Glial Signaling in Pathological Pain: Distinct Roles of Spinal Microglia and Astrocytes. Neuroscience Bulletin 2018, 34: 98-108.
[10] Moore C,Gupta R, Jordt SE, Chen Y, Liedtke WB. Regulation of Pain and Itch by TRP Channels. Neuroscience Bulletin 2018, 34: 120-142.
[11] AndersonM, Zheng Q, Dong XZ. Investigation of Pain Mechanisms by Calcium Imaging Approaches. Neuroscience Bulletin 2018, 34: 194-199.
[12] Xie RG,Gao YJ, Park CK, Lu N, Luo C, Wang WT, etal. Spinal CCL2 Promotes Central Sensitization, Long-Term Potentiation, and Inflammatory Pain via CCR2: Further Insights into Molecular, Synaptic, and Cellular Mechanisms. Neuroscience Bulletin 2018, 34: 13-21.
[13] Duan B,Cheng LZ, Ma QF. Spinal Circuits Transmitting Mechanical Pain and Itch.Neuroscience Bulletin 2018, 34: 186-193.
[14] Kong XP,Wei JR, Wang DY, Zhu XJ, Zhou YL, Wang SS,et al. Upregulation of Spinal Voltage-Dependent Anion Channel 1 Contributes to Bone Cancer Pain Hypersensitivity in Rats. Neuroscience Bulletin 2017, 33:711-721.
[15] Zhou FM,Cheng RX, Wang S, Huang Y, Gao YJ, Zhou Y,et al. Antioxidants Attenuate Acute and Chronic Itch: Peripheral and Central Mechanisms of Oxidative Stress in Pruritus. Neuroscience Bulletin 2017,33: 423-435.